Download Anabolic steroids: and other performance-enhancing drugs by Patrick Lenehan PDF

By Patrick Lenehan

An exceptionally topical overview of the way anabolic steroids are taking part in an more and more prominant half in non-competitive activity. Brings jointly the most important matters in a lucid layout that might attract a wide variety of scholars and execs.

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1976). ALT and AST are also produced by muscle, heart and bone and so the interpretation of liver function assays can be difficult to analyse, as elevations in these liver enzymes can result after exercise or after tissue damage such as might be caused by injecting steroids. If the raised levels are attributable to AS then they are usually reversible after stopping steroid use or are transient even if AS use continues (Lamb, 1984). It has been suggested that future studies of AS use in athletes should monitor the enzymes lactate dehydrogenase (LDH) and alkaline phosphate (ALP), which are more specific to the function of the liver (Kibble and Ross, 1987).

11 Legal status of anabolic steroids Anabolic steroids have different legal statuses in different parts of the world. In Britain, AS became subject to the control of The Misuse of Drugs Act on 1 November 1996. They are now categorized as class C drugs and possible penalties for offences are 5 years imprisonment, or an unlimited fine, or both. These restrictions mean that although it is legal to possess AS in quantities for personal use, it is illegal to produce, to supply, or to possess, import or export with intent to supply these substances, without the authority to do so.

Central prostatic volume was increased by 20% and the central prostatic volume/peripheral prostatic volume ratio was increased by 77%. Serum changes showed a 47% decrease in sex hormone binding globulin, prostatic acid phosphatase was increased by 26% and LH, FSH and total testosterone were significantly reduced. Prostate specific antigen was within normal limits. , 1996). There may also be an association with prostatic cancer due to stimulation from excess androgens resulting from AS use (Schally and Comaru-Schally, 1987).

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